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The mice displayed symptoms similar to human fainting, including rapid pupil dilation and rolling eyes. This discovery shed light on the underlying mechanisms of fainting bouts.Vineet Augustine, study co-author, and a neuroscientist at the University of California, San Diego stated: The research enabled scientists to devise tools for understanding the connection between the brain and the heart. Using single-cell RNA sequencing analysis of the nodose ganglion, a part of the vagus nerve that links the brain to various organs, including the heart.These sensory neurons, known as NPY2R VSNs, differ from other branches of the vagus nerve that connect to organs like the lungs or gut.
Dijk further explained that these nerve cells begin to die after roughly two to five minutes without oxygen, however, syncope commonly lasts less than 60 seconds.Additionally, researchers employed electrodes to record brain activity in mice during the fainting episodes. The analysis showed that most areas of the brain demonstrated decreased activity with the exception of a specific region – the periventricular zone .
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Source: IntEngineering - 🏆 287. / 63 Read more »
Source: IntEngineering - 🏆 287. / 63 Read more »
Source: IntEngineering - 🏆 287. / 63 Read more »
Source: IntEngineering - 🏆 287. / 63 Read more »
Source: IntEngineering - 🏆 287. / 63 Read more »
Source: IntEngineering - 🏆 287. / 63 Read more »